Further Evaluating the Relationship Between Adverse Childhood Experiences, Antisocial Behavior, and Violent Victimization: A Sibling-Comparison Analysis

Date01 January 2020
AuthorEric J. Connolly
Published date01 January 2020
DOI10.1177/1541204019833145
Subject MatterArticles
Article
Further Evaluating the
Relationship Between
Adverse Childhood Experiences,
Antisocial Behavior, and Violent
Victimization: A Sibling-Comparison
Analysis
Eric J. Connolly
1
Abstract
A developing line of research suggests that adverse childhood experiences (ACEs) increase the
risk for antisocial behavior and future victimization. However, the mechanisms that underlie this
association remain largely speculative. To address this gap in the existing body of research, data on
full siblings from a large population-based sample of youth were analyzed to evaluate the direct
effect of ACEs on child antisocial behavior, adolescent delinquency, and young adult violent vic-
timization after controlling for familial confounders. Traditional between-family analyses revealed
that ACEs were significantly associated with higher levels of childhood antisocial behavior, ado-
lescent delinquent behavior, and risk for violent crime victimization. After controlling for
unmeasured common genetic and shared environmental confounds using fixed-effect sibling
comparisons, siblings exposed to more ACEs did not demonstrate higher levels of antisocial
behavior, delinquent behavior, or risk for future victimization. The implications of these results for
future ACEs research are discussed.
Keywords
adverse childhood experiences, antisocial behavior, violent victimization, familial confounds, family-
based research designs, NLSY
1
Department of Criminal Justice and Criminology, College of Criminal Justice, Sam Houston State University, Huntsville,
TX, USA
Corresponding Author:
Eric J. Connolly, Department of Criminal Justice and Criminology, College of Criminal Justice, Sam Houston State University,
Huntsville, TX 77340, USA.
Email: ejc025@shsu.edu
Youth Violence and JuvenileJustice
2020, Vol. 18(1) 3-23
ªThe Author(s) 2019
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DOI: 10.1177/1541204019833145
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We may be through with the past, but the past is not through with us.
Evans (1946)
Research regarding the effect of adversechildhood experiences (ACEs)on later-life health and human
development has grown at a rapid rate over the past decade. This growing body of research can be
traced back to the landmark Center for Disease Control-Kaiser ACE study conducted by Felitti and
colleagues (1998), which examined the impact of ACEs on health-related outcomes in adulthood.
Results from Felitti et al. (1998) showed, for the first time, that ACEs increase risk for cancer, heart
disease, lung disease, liver disease, and early death. Recently, criminologists have begun to apply the
ACE framework to the study of individual differences in antisocial behavior and criminal victimiza-
tion. Several studies have documented a dose–response relationship between ACEs and a wide range
of antisocial behaviors including childhood externalizing problems (Hunt, Slack, & Berger, 2017),
preadolescentdelinquent behavior (Hambrick,Rubens, Brawner, & Taussig,2017), violent delinquent
behavior (Fox,Perez, Cass, Baglivio, & Epps, 2015), juvenile arrest(Fagan & Novak, 2017), juvenile
offending traject ories (Baglivio , Wolff, Piquero, & Epps, 2 015), juvenile recid ivism (Wolff & Bagli-
vio, 2016; Wolff,Baglivio, & Piquero, 2017), and life-course offending (Craig,Piquero, Farrington, &
Ttofi, 2017). Studies also report a positive associationbetween ACEs and future victimization (Ports,
Ford, & Merrick, 2016; Widom, Czaja, & Dutton, 2008). Taken together, accumulated evidence
indicates that while individuals may be done with ACEs, such experiences may not be done with
them and continue to exert an effect on their health and behavior.
While evidence of a robust association between ACEs, antisocial behavior, and future victimiza-
tion is mounting, there are some important limitations that warrant further examination. First,
evidence from contemporary research on ACEs within criminology is largely based on data from
at-risk samples of delinquent youth, making it difficult to establish whether findings are general-
izable to more representative youth samples. As a result, it is currently unknown whether reported
results can be generalized to nonoffending populations in order to inform evidence-based interven-
tion/prevention programing efforts. Second, while existing research has used a range of multivariate
statistical techniques to control for measurable confounds, no research within criminology has used
a quasi-experimental, genetically informed research design to control for unobservable genetic and
environmental confounds that may partly explain the relationship between ACEs, antisocial beha-
vior, and future victimization. Of primary theoretical significance to this point, a long line of
quantitative behavioral genetic research has now shown that correlations between environmental
exposure and individual’s genetic risk for antisocial behavior and victimization are pervasive across
the life course (Barnes & Beaver, 2012; Connolly & Beaver, 2015; D’Onofrio et al., 2016; Jaffee &
Price, 2012; Kendler & Baker, 2007; Schwartz, Solomon, & Valgardson, 2017). A failure to control
for familial confounds may therefore inflate associations between early-life exposures (i.e., ACEs)
and life outcomes (i.e., antisocial behavior and victimization). Thus, genetically informed research
designs are greatly needed in criminology to allow researchers to move from identifying risks to
examining mechanisms (genetic and/or environmental) by which a risk factor may potentially cause
future antisocial behavior or risk for victimization.
With this in mind, the current study seeks to address the abovementioned limitations by
(1) analyzing longitudinal data from a population-based sample of youth from the United States
to examine the prevalence of ACEs and compare rates to previous research and (2) using traditional
between-family analyses and sibling-comparison analyses to help disentangle co-occurring genetic
and environmental processes between ACEs, antisocial behavior in childhood, delinquent behavior
in adolescence, and violent victimization in young adulthood. The overall rationale for this study is
to use a rigorous quasi-experimental method, such as the sibling-comparison design, to help specify
the processes behind commonly observed associations between ACEs, antisocial behavior,
4Youth Violence and Juvenile Justice 18(1)

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